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It was previously believed that the intermittent increase in blood pressure that is caused by heavy lifting was not sufficient to elicit concentric left ventricular hypertrophy (CLVH). chemistry of steroids, muscle men gallery, weight loss steroids, steroid types, pictures of anabolic steroid side effects, topical steroids, steroid withdrawal, teenage bodybuilding, steroid rage, steroid use facts Steroid rage. Any evidence of CLVH in strength athletes or bodybuilders was seen as a sign of anabolic steroid use. During heavy lifting, systemic blood pressure is increased from what is called the valsalva maneuver. It is simply the act of forceful expiration with the mouth and nose closed producing a "bearing down" on the abdomen. chemistry of steroids, muscle men gallery, weight loss steroids, steroid types, pictures of anabolic steroid side effects, topical steroids, steroid withdrawal, teenage bodybuilding, steroid rage, steroid use facts Steroid use facts. Most people do this during heavy lifts such as squats or deadlifts. Pressure also increases due to blood vessels being occluded by contracting muscles. It should be noted that the LVH seen in bodybuilders and power lifters is called "concentric left ventricular hypertrophy", meaning that it is the result of contracting against acute increased systemic pressure, and is not considered pathological (i. chemistry of steroids, muscle men gallery, weight loss steroids, steroid types, pictures of anabolic steroid side effects, topical steroids, steroid withdrawal, teenage bodybuilding, steroid rage, steroid use facts Weight loss steroids. e. unhealthy). "Eccentric" LVH is caused by constant increases of blood pressure, not as a result of the valsalva maneuver but instead due to clinical hypertension that forces the ventricle to expand against resistance. AAS further exacerbate the effects of lifting on the heart. AASs cause anabolism in heart muscle, at times increasing left ventricular wall thickness to 16mm (11mm is considered normal). 4 However, LVH caused by resistance training either alone or in conjunction with AAS has yet to result in diastolic dysfunction, or in other words, there is yet no evidence that this thickening of the ventricular wall is pathologic. Treatment optionsUpon cessation of high intensity resistance exercise and obviously AAS use, ventricular wall thickness returns to within normal ranges as long as hypertension unrelated to lifting is not present. There are no treatment suggestions for LVH caused by resistance training with or without the use of AAS. HepaticAbnormal liver function tests are another common reported side effect of AAS use. However the main concern, or danger, is the appearance of peliosis hepatitis (An abnormal condition characterized by the occurrence of small blood-filled cystic lesions throughout the liver) or liver tumors. The occurrence of these serious problems is rare. Almost all reported cases are associated with heavy 17alpha-alkylated anabolic steroid use and occur in patients with pre-existing medical conditions. 5As with left ventricular hypertrophy, physicians unfamiliar with the effects of resistance training often misdiagnose abnormal liver function tests. A recent study from the Department of Medicine, University of North Texas Health Science Center had this to say:Numerous reports have noted "hepatic" dysfunction secondary to anabolic steroid use based on elevated serum aminotransferase levels. The authors' objective was to assess whether primary care physicians accurately distinguish between anabolic steroid-induced hepatotoxicity and serum aminotransferase elevations that are secondary to acute rhabdomyolysis resulting from intense resistance training. Surveys were sent to physicians listed as practicing family medicine or sports medicine in the yellow pages of seven metropolitan areas. Physicians were asked to provide a differential diagnosis for a 28-year-old, anabolic steroid-using male bodybuilder with an abnormal serum chemistry profile. The blood chemistries showed elevated aspartate aminotransferase (AST), alanine aminotransferase (ALT), and creatine kinase (CK) levels, and normal gamma-glutamyltransferase (GGT) levels. In the physician survey (n = 84 responses), 56% failed to mention muscle damage or muscle disease as a potential diagnosis, despite the markedly elevated CK level of the patient.

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